Sunday, January 11, 2015

Docket No. APHIS-2014-0107 Bovine Spongiform Encephalopathy; Importation of Animals and Animal Products Singeltary Submission

Agency Information Collection Activities; Proposals, Submissions, and Approvals: Bovine Spongiform Encephalopathy; Importation of Animals and Animal Products.

 

This Notice document was issued by the Animal and Plant Health Inspection Service (APHIS)

 

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Show agency attachment(s) DEPARTMENT OF AGRICULTURE

 

Animal and Plant Health Inspection Service

 

[Docket No. APHIS-2014-0107]

 

Notice of Request for Revision to and Extension of Approval of an Information Collection; Bovine Spongiform Encephalopathy; Importation of Animals and Animal ProductsAgencyAnimal and Plant Health Inspection Service, USDA.

 

Action Revision to and extension of approval of an information collection; comment request.

 

Summary In accordance with the Paperwork Reduction Act of 1995, this notice announces the Animal and Plant Health Inspection Service's intention to request a revision to and extension of approval of an information collection associated with the regulations for the importation of animals and animal products and byproducts to protect against the introduction of bovine spongiform encephalopathy into the United States.

 

Dates We will consider all comments that we receive on or before March 2, 2015.

 

Addresses You may submit comments by either of the following methods:

 


 

•Postal Mail/Commercial Delivery: Send your comment to Docket No. APHIS-2014-0107, Regulatory Analysis and Development, PPD, APHIS, Station 3A-03.8, 4700 River Road Unit 118, Riverdale, MD 20737-1238. Supporting documents and any comments we receive on this docket may be viewed at http://www.regulations.gov/#!docketDetail;D=APHIS-2014-0107 or in our reading room, which is located in Room 1141 of the USDA South Building, 14th Street and Independence Avenue SW., Washington, DC. Normal reading room hours are 8 a.m. to 4:30 p.m., Monday through Friday, except holidays. To be sure someone is there to help you, please call (202) 799-7039 before coming.

 

For Further Information Contact For information on the regulations for the importation of animals and animal products and byproducts to prevent the introduction of bovine spongiform encephalopathy into the United States, contact Dr. Langston Hull, Senior Staff Veterinarian, Veterinary Services, APHIS, 4700 River Road, Unit 39, Riverdale, MD 20737; (301) 851-3363. For copies of more detailed information on the information collection, contact Ms. Kimberly Hardy, APHIS' Information Collection Coordinator, at (301) 851-2727.

 

Supplementary Information Title: Bovine Spongiform Encephalopathy; Importation of Animals and Animal Products.

 

OMB Control Number: 0579-0234.

 

Type of Request: Revision to and extension of approval of an information collection.

 

Abstract: Under the Animal Health Protection Act (7 U.S.C. 8301 et seq.), the Animal and Plant Health Inspection Service of the U.S. Department of Agriculture regulates the importation of animals and animal products into the United States to guard against the introduction of animal diseases. The regulations in 9 CFR parts 93, 94, 95, and 96 (referred to below as the regulations) govern the importation of certain animals, birds, poultry, meat, other animal products and byproducts, hay, and straw into the United States in order to prevent the introduction of animal diseases, including bovine spongiform encephalopathy (BSE), a chronic degenerative disease affecting the central nervous system of cattle.

 

To help ensure that BSE is not introduced into the United States, the regulations place specified conditions on the importation of certain live ruminants and ruminant products and byproducts. These requirements necessitate the use of several information collection activities, including Veterinary Services (VS) Form 16-3, permit application; certification statements for the importation of ruminants and ruminant products; certificate for inedible processed animal origin materials and products from BSE-free regions; cooperative service agreements with foreign facilities that process and store regulated materials and products destined for importation into the United States; VS Form 17-33, Animals Imported for Immediate Slaughter; the placing of seals on conveyances from the exporting region; agreement with slaughter facilities on use of seals on conveyances transporting animals from BSE minimal-risk regions; notification regarding conditions of sealed shipments; and notification of designated individuals authorized to break seals.

 

In addition to the above information collection activities, we are adding VS Form 17-130, Ruminants Imported to Designated/Approved Feedlots; and VS Form 1-27, Permit for Movement of Restricted Animals. As a result of adding these two activities and the increase in importations from Canada, the estimated annual number of responses has increased by 110,463, and the estimated total annual burden on respondents has increased by 160,983 hours.

 

We are asking the Office of Management and Budget (OMB) to approve our use of these information collection activities, as described, for an additional 3 years.

 

The purpose of this notice is to solicit comments from the public (as well as affected agencies) concerning our information collection. These comments will help us:

 

(1) Evaluate whether the collection of information is necessary for the proper performance of the functions of the Agency, including whether the information will have practical utility;

 

(2) Evaluate the accuracy of our estimate of the burden of the collection of information, including the validity of the methodology and assumptions used;

 

(3) Enhance the quality, utility, and clarity of the information to be collected; and

 

(4) Minimize the burden of the collection of information on those who are to respond, through use, as appropriate, of automated, electronic, mechanical, and other collection technologies; e.g., permitting electronic submission of responses.

 

Estimate of burden: The public reporting burden for this collection of information is estimated to average 1 hour per response.

 

Respondents: Herd owners, U.S. importers of regulated animal products, salaried veterinarians in BSE-free regions and BSE-affected regions, foreign exporters of processed animal protein and other regulated materials and products, accredited veterinarians, feedlot managers, and slaughter facility managers.

 

Estimated annual number of respondents: 4,500.

 

Estimated annual number of responses per respondent: 52.

 

Estimated annual number of responses: 235,752.

 

Estimated total annual burden on respondents: 231,307 hours. (Due to averaging, the total annual burden hours may not equal the product of the annual number of responses multiplied by the reporting burden per response.)

 

All responses to this notice will be summarized and included in the request for OMB approval. All comments will also become a matter of public record.

 

Done in Washington, DC, this 22nd day of December 2014. Kevin Shea, Administrator, Animal and Plant Health Inspection Service. [FR Doc. 2014-30501 Filed 12-29-14; 8:45 am] BILLING CODE 3410-34-P

 


 


 


 

submit here ;

 


 

VS.Live.Animal.Import.Export@aphis.usda.gov;

 

 

Docket No. APHIS-2014-0107 Bovine Spongiform Encephalopathy; Importation of Animals and Animal Products Singeltary Submission ;

 

 

Greetings APHIS et al,

 

I would kindly like to comment on the following docket ;

 

Docket No. APHIS-2014-0107 Bovine Spongiform Encephalopathy; Importation of Animals and Animal Products

 

My comment as follows ;

 

This would be comical if not so serious. the USDA and the OIE shoved this BSE Minimal Risk Region MRR policy, the legal trading of mad cow type TSE prion disease around the globe, down the throats of every Country around the world, after that fateful day December 23, 2003, when the USDA et al lost it’s BSE FREE gold card. then they went and changed science, and now APHIS/USDA et al are worried about getting BSE. r e a l l y $ 

 

I believe that there is more risk to the world from Transmissible Spongiform Encephalopathy TSE prion aka mad cow type disease now, coming from the United States and all of North America, than there is risk coming to the USA and North America, from other Countries. I am NOT saying I don’t think there is any risk for the BSE type TSE prion coming from other Countries, I am just saying that in 2015, why is the APHIS/USDA/FSIS/FDA still ignoring these present mad cow risk factors in North America like they are not here?

 

North America has more strains of TSE prion disease, in more species (excluding zoo animals in the early BSE days, and excluding the Feline TSE and or Canine TSE, because they don’t look, and yes, there has been documented evidence and scientific studies, and DEFRA Hound study, that shows the canine spongiform encephalopathy is very possible, if it has not already happened, just not documented), then any other Country in the world. Mink TME, Deer Elk cervid CWD (multiple strains), cBSE cattle, atypical L-type BSE cattle, atypical H-type BSE cattle, atyical HG type BSE cow (the only cow documented in the world to date with this strain), typical sheep goat Scrapie (multiple strains), and the atypical Nor-98 Scrapie, which has been linked to sporadic CJD, Nor-98 atypical Scrapie has spread from coast to coast. sporadic CJD on the rise, with different strains mounting, victims becoming younger, with the latest nvCJD human mad cow case being documented in Texas again, this case, NOT LINKED TO EUROPEAN TRAVEL CDC.

 

typical BSE can propagate as nvCJD and or sporadic CJD (Collinge et al), and sporadic CJD has now been linked to atypical BSE, Scrapie and atypical Scrapie, and scientist are very concerned with CWD TSE prion in the Cervid populations. in my opinion, the BSE MRR policy, which overtook the BSE GBR risk assessments for each country, and then made BSE confirmed countries legal to trade mad cow disease, which was all brought forth AFTER that fateful day December 23, 2003, when the USA lost it’s gold card i.e. BSE FREE status, that’s the day it all started. once the BSE MRR policy was shoved down every countries throat by USDA inc and the OIE, then the legal trading of Scrapie was validated to be a legal trading commodity, also shoved through by the USDA inc and the OIE, the world then lost 30 years of attempted eradication of the BSE TSE prion disease typical and atypical strains, and the BSE TSE Prion aka mad cow type disease was thus made a legal trading commodity, like it or not. it’s all about money now folks, trade, to hell with human health with a slow incubating disease, that is 100% fatal once clinical, and forget the fact of exposure, sub-clinical infection, and friendly fire there from i.e. iatrogenic TSE prion disease, the pass it forward mode of the TSE PRION aka mad cow type disease. it’s all going to be sporadic CJD or sporadic ffi, or sporadic gss, or now the infamous VPSPr. ...problem solved $$$

 

the USDA/APHIS/FSIS/FDA triple mad cow BSE firewall, well, that was nothing but ink on paper.

 

for this very reason I believe the BSE MRR policy is a total failure, and that this policy should be immediately withdrawn, and set back in place the BSE GBR Risk Assessments, with the BSE GBR risk assessments set up to monitor all TSE PRION disease in all species of animals, and that the BSE GBR risk assessments be made stronger than before.

 

let’s start with the recent notice that beef from Ireland will be coming to America.

 

Ireland confirmed around 1655 cases of mad cow disease. with the highest year confirming about 333 cases in 2002, with numbers of BSE confirmed cases dropping from that point on, to a documentation of 1 confirmed case in 2013, to date. a drastic decrease in the feeding of cows to cows i.e. the ruminant mad cow feed ban, and the enforcement of that ban, has drastically reduced the number of BSE cases in Europe, minus a few BAB’s or BARB’s. a far cry from the USDA FDA triple BSE firewall, which was nothing more than ink on paper, where in 2007, in one week recall alone, some 10 MILLION POUNDS OF BANNED POTENTIAL MAD COW FEED WENT OUT INTO COMMERCE IN THE USA. this is 10 years post feed ban. in my honest opinion, due to the blatant cover up of BSE TSE prion aka mad cow disease in the USA, we still have no clue as to the true number of cases of BSE mad cow disease in the USA or North America as a whole. ...just saying.

 

Number of reported cases of bovine spongiform encephalopathy (BSE) in farmed cattle worldwide* (excluding the United Kingdom)

 

Country/Year

 

snip...please see ;

 


 

2007

 

Date: March 21, 2007 at 2:27 pm PST RECALLS AND FIELD CORRECTIONS: VETERINARY MEDICINES -- CLASS II PRODUCT Bulk cattle feed made with recalled Darling's 85% Blood Meal, Flash Dried, Recall # V-024-2007 CODE Cattle feed delivered between 01/12/2007 and 01/26/2007 RECALLING FIRM/MANUFACTURER Pfeiffer, Arno, Inc, Greenbush, WI. by conversation on February 5, 2007.

 

Firm initiated recall is ongoing. REASON Blood meal used to make cattle feed was recalled because it was cross- contaminated with prohibited bovine meat and bone meal that had been manufactured on common equipment and labeling did not bear cautionary BSE statement.

 

VOLUME OF PRODUCT IN COMMERCE 42,090 lbs. DISTRIBUTION WI

 

___________________________________

 

PRODUCT Custom dairy premix products: MNM ALL PURPOSE Pellet, HILLSIDE/CDL Prot- Buffer Meal, LEE, M.-CLOSE UP PX Pellet, HIGH DESERT/ GHC LACT Meal, TATARKA, M CUST PROT Meal, SUNRIDGE/CDL PROTEIN Blend, LOURENZO, K PVM DAIRY Meal, DOUBLE B DAIRY/GHC LAC Mineral, WEST PIONT/GHC CLOSEUP Mineral, WEST POINT/GHC LACT Meal, JENKS, J/COMPASS PROTEIN Meal, COPPINI - 8# SPECIAL DAIRY Mix, GULICK, L-LACT Meal (Bulk), TRIPLE J - PROTEIN/LACTATION, ROCK CREEK/GHC MILK Mineral, BETTENCOURT/GHC S.SIDE MK-MN, BETTENCOURT #1/GHC MILK MINR, V&C DAIRY/GHC LACT Meal, VEENSTRA, F/GHC LACT Meal, SMUTNY, A- BYPASS ML W/SMARTA, Recall # V-025-2007 CODE The firm does not utilize a code - only shipping documentation with commodity and weights identified. RECALLING FIRM/MANUFACTURER Rangen, Inc, Buhl, ID, by letters on February 13 and 14, 2007.

 

Firm initiated recall is complete. REASON Products manufactured from bulk feed containing blood meal that was cross contaminated with prohibited meat and bone meal and the labeling did not bear cautionary BSE statement.

 

VOLUME OF PRODUCT IN COMMERCE 9,997,976 lbs. DISTRIBUTION ID and NV

 

END OF ENFORCEMENT REPORT FOR MARCH 21, 2007

 


 

Tuesday, December 23, 2014

 

FDA PART 589 -- SUBSTANCES PROHIBITED FROM USE IN ANIMAL FOOD OR FEED VIOLATIONS OFFICIAL ACTION INDICATED OAI UPDATE DECEMBER 2014 BSE TSE PRION

 


 

Sunday, December 15, 2013

 

FDA PART 589 -- SUBSTANCES PROHIBITED FROM USE IN ANIMAL FOOD OR FEED VIOLATIONS OFFICIAL ACTION INDICATED OIA UPDATE DECEMBER 2013 UPDATE

 


 

Thursday, July 24, 2014

 

*** Protocol for further laboratory investigations into the distribution of infectivity of Atypical BSE SCIENTIFIC REPORT OF EFSA New protocol for Atypical BSE investigations

 


 

*** Singeltary reply ; Molecular, Biochemical and Genetic Characteristics of BSE in Canada Singeltary reply ;

 


 

Thursday, November 18, 2010

 

UNITED STATES OF AMERICA VS GALEN J. NIEHUES FAKED MAD COW FEED TEST ON 92 BSE INSPECTION REPORTS FOR APPROXIMATELY 100 CATTLE OPERATIONS

 

Dustin Douglass was indicted and charged with making a fraudulent application to the VA, in an effort to obtain benefits from injuries Douglas represented he suffered while deployed in Iraq. Based on his application, the VA provided benefits totaling $22,148.53. Douglass claimed he suffered various injuries and illnesses as a result of his service in combat. The investigation revealed Douglass had, in fact, been deployed to Iraq, but had served as a computer specialist, had never been in combat, and did not suffer the service-related injuries and illnesses he claimed to have suffered. Douglass was placed on supervised release for 3 years, and required to pay $22,148.53 in restitution. Galen Niehues, an inspector for the Nebraska Department of Agriculture, (NDA), was convicted of mail fraud for submitting falsified reports to his employer concerning inspections he was supposed to perform at Nebraska cattle operations. Niehues was tasked with performing inspections of Nebraska ranches, cattle and feed for the presence of neurological diseases in cattle including Bovine Spongiform Encephalopathy (BSE), also known as “Mad Cow Disease”. Niehues was to identify cattle producers, perform on-site inspections of the farm sites and cattle operations, ask producers specific questions about feed, and take samples of the feed. Niehues was to then submit feed samples for laboratory analysis, and complete reports of his inspections and submit them to the NDA and to the Federal Food and Drug Administration (FDA). An investigation by the FDA and NDA revealed Niehues had fabricated approximately 100 BSE inspections and inspection reports. When confronted, Niehues admitted his reports were fraudulent, and that had fabricated the reports and feed samples he submitted to the NDA. Niehues received a sentence of 5 years probation, a 3-year term of supervised release, and was required to pay $42,812.10 in restitution.

 


 


 

The 2004 enhanced BSE surveillance program was so flawed, that one of the top TSE prion Scientist for the CDC, Dr. Paul Brown stated ; Brown, who is preparing a scientific paper based on the latest two mad cow cases to estimate the maximum number of infected cows that occurred in the United States, said he has "absolutely no confidence in USDA tests before one year ago" because of the agency's reluctance to retest the Texas cow that initially tested positive.

 

see ;

 


 

CDC - Bovine Spongiform Encephalopathy and Variant Creutzfeldt ... Dr. Paul Brown is Senior Research Scientist in the Laboratory of Central Nervous System ... Address for correspondence: Paul Brown, Building 36, Room 4A-05, ...

 


 

PAUL BROWN COMMENT TO ME ON THIS ISSUE

 

Tuesday, September 12, 2006 11:10 AM

 

"Actually, Terry, I have been critical of the USDA handling of the mad cow issue for some years, and with Linda Detwiler and others sent lengthy detailed critiques and recommendations to both the USDA and the Canadian Food Agency."

 

OR, what the Honorable Phyllis Fong of the OIG found ;

 

Finding 2 Inherent Challenges in Identifying and Testing High-Risk Cattle Still Remain

 


 

IT is of my opinion, that the OIE and the USDA et al, are the soul reason, and responsible parties, for Transmissible Spongiform Encephalopathy TSE prion diseases, including typical and atypical BSE, typical and atypical Scrapie, and all strains of CWD, and human TSE there from, spreading around the globe. I have lost all confidence of this organization as a regulatory authority on animal disease, and consider it nothing more than a National Trading Brokerage for all strains of animal TSE, just to satisfy there commodity. AS i said before, OIE should hang up there jock strap now, since it appears they will buckle every time a country makes some political hay about trade protocol, commodities and futures. IF they are not going to be science based, they should do everyone a favor and dissolve there organization. JUST because of low documented human body count with nvCJD and the long incubation periods, the lack of sound science being replaced by political and corporate science in relations with the fact that science has now linked some sporadic CJD with atypical BSE and atypical scrapie, and the very real threat of CWD being zoonosis, I believed the O.I.E. has failed terribly and again, I call for this organization to be dissolved. ...

 

Monday, May 05, 2014

 

Member Country details for listing OIE CWD 2013 against the criteria of Article 1.2.2., the Code Commission recommends consideration for listing

 


 

Friday, December 5, 2014

 

SPECIAL ALERT The OIE recommends strengthening animal disease surveillance worldwide

 


 

IN A NUT SHELL ; (Adopted by the International Committee of the OIE on 23 May 2006) 11. Information published by the OIE is derived from appropriate declarations made by the official Veterinary Services of Member Countries. The OIE is not responsible for inaccurate publication of country disease status based on inaccurate information or changes in epidemiological status or other significant events that were not promptly reported to the Central Bureau,

 


 

Evidence for zoonotic potential of ovine scrapie prions

 

Hervé Cassard,1, n1 Juan-Maria Torres,2, n1 Caroline Lacroux,1, Jean-Yves Douet,1, Sylvie L. Benestad,3, Frédéric Lantier,4, Séverine Lugan,1, Isabelle Lantier,4, Pierrette Costes,1, Naima Aron,1, Fabienne Reine,5, Laetitia Herzog,5, Juan-Carlos Espinosa,2, Vincent Beringue5, & Olivier Andréoletti1, Affiliations Contributions Corresponding author Journal name: Nature Communications Volume: 5, Article number: 5821 DOI: doi:10.1038/ncomms6821 Received 07 August 2014 Accepted 10 November 2014 Published 16 December 2014 Article tools Citation Reprints Rights & permissions Article metrics

 

Abstract

 

Although Bovine Spongiform Encephalopathy (BSE) is the cause of variant Creutzfeldt Jakob disease (vCJD) in humans, the zoonotic potential of scrapie prions remains unknown. Mice genetically engineered to overexpress the human ​prion protein (tgHu) have emerged as highly relevant models for gauging the capacity of prions to transmit to humans. These models can propagate human prions without any apparent transmission barrier and have been used used to confirm the zoonotic ability of BSE. Here we show that a panel of sheep scrapie prions transmit to several tgHu mice models with an efficiency comparable to that of cattle BSE. The serial transmission of different scrapie isolates in these mice led to the propagation of prions that are phenotypically identical to those causing sporadic CJD (sCJD) in humans. These results demonstrate that scrapie prions have a zoonotic potential and raise new questions about the possible link between animal and human prions.

 

Subject terms: Biological sciences• Medical research At a glance

 


 

Suspect symptoms

 

What if you can catch old-fashioned CJD by eating meat from a sheep infected with scrapie?

 

28 Mar 01 Most doctors believe that sCJD is caused by a prion protein deforming by chance into a killer. But Singeltary thinks otherwise. He is one of a number of campaigners who say that some sCJD, like the variant CJD related to BSE, is caused by eating meat from infected animals. Their suspicions have focused on sheep carrying scrapie, a BSE-like disease that is widespread in flocks across Europe and North America.

 

Now scientists in France have stumbled across new evidence that adds weight to the campaigners' fears. To their complete surprise, the researchers found that one strain of scrapie causes the same brain damage in mice as sCJD.

 

"This means we cannot rule out that at least some sCJD may be caused by some strains of scrapie," says team member Jean-Philippe Deslys of the French Atomic Energy Commission's medical research laboratory in Fontenay-aux-Roses, south-west of Paris. Hans Kretschmar of the University of Göttingen, who coordinates CJD surveillance in Germany, is so concerned by the findings that he now wants to trawl back through past sCJD cases to see if any might have been caused by eating infected mutton or lamb...

 

2001

 

Suspect symptoms

 

What if you can catch old-fashioned CJD by eating meat from a sheep infected with scrapie?

 

28 Mar 01

 

Like lambs to the slaughter

 

31 March 2001

 

by Debora MacKenzie Magazine issue 2284.

 

FOUR years ago, Terry Singeltary watched his mother die horribly from a degenerative brain disease. Doctors told him it was Alzheimer's, but Singeltary was suspicious. The diagnosis didn't fit her violent symptoms, and he demanded an autopsy. It showed she had died of sporadic Creutzfeldt-Jakob disease.

 

Most doctors believe that sCJD is caused by a prion protein deforming by chance into a killer. But Singeltary thinks otherwise. He is one of a number of campaigners who say that some sCJD, like the variant CJD related to BSE, is caused by eating meat from infected animals. Their suspicions have focused on sheep carrying scrapie, a BSE-like disease that is widespread in flocks across Europe and North America.

 

Now scientists in France have stumbled across new evidence that adds weight to the campaigners' fears. To their complete surprise, the researchers found that one strain of scrapie causes the same brain damage in mice as sCJD.

 

"This means we cannot rule out that at least some sCJD may be caused by some strains of scrapie," says team member Jean-Philippe Deslys of the French Atomic Energy Commission's medical research laboratory in Fontenay-aux-Roses, south-west of Paris. Hans Kretschmar of the University of Göttingen, who coordinates CJD surveillance in Germany, is so concerned by the findings that he now wants to trawl back through past sCJD cases to see if any might have been caused by eating infected mutton or lamb.

 

Scrapie has been around for centuries and until now there has been no evidence that it poses a risk to human health. But if the French finding means that scrapie can cause sCJD in people, countries around the world may have overlooked a CJD crisis to rival that caused by BSE.

 

Deslys and colleagues were originally studying vCJD, not sCJD. They injected the brains of macaque monkeys with brain from BSE cattle, and from French and British vCJD patients. The brain damage and clinical symptoms in the monkeys were the same for all three. Mice injected with the original sets of brain tissue or with infected monkey brain also developed the same symptoms.

 

As a control experiment, the team also injected mice with brain tissue from people and animals with other prion diseases: a French case of sCJD; a French patient who caught sCJD from human-derived growth hormone; sheep with a French strain of scrapie; and mice carrying a prion derived from an American scrapie strain. As expected, they all affected the brain in a different way from BSE and vCJD. But while the American strain of scrapie caused different damage from sCJD, the French strain produced exactly the same pathology.

 

"The main evidence that scrapie does not affect humans has been epidemiology," says Moira Bruce of the neuropathogenesis unit of the Institute for Animal Health in Edinburgh, who was a member of the same team as Deslys. "You see about the same incidence of the disease everywhere, whether or not there are many sheep, and in countries such as New Zealand with no scrapie." In the only previous comparisons of sCJD and scrapie in mice, Bruce found they were dissimilar.

 

But there are more than 20 strains of scrapie, and six of sCJD. "You would not necessarily see a relationship between the two with epidemiology if only some strains affect only some people," says Deslys. Bruce is cautious about the mouse results, but agrees they require further investigation. Other trials of scrapie and sCJD in mice, she says, are in progress.

 

People can have three different genetic variations of the human prion protein, and each type of protein can fold up two different ways. Kretschmar has found that these six combinations correspond to six clinical types of sCJD: each type of normal prion produces a particular pathology when it spontaneously deforms to produce sCJD.

 

But if these proteins deform because of infection with a disease-causing prion, the relationship between pathology and prion type should be different, as it is in vCJD. "If we look at brain samples from sporadic CJD cases and find some that do not fit the pattern," says Kretschmar, "that could mean they were caused by infection."

 

There are 250 deaths per year from sCJD in the US, and a similar incidence elsewhere. Singeltary and other US activists think that some of these people died after eating contaminated meat or "nutritional" pills containing dried animal brain. Governments will have a hard time facing activists like Singeltary if it turns out that some sCJD isn't as spontaneous as doctors have insisted.

 

Deslys's work on macaques also provides further proof that the human disease vCJD is caused by BSE. And the experiments showed that vCJD is much more virulent to primates than BSE, even when injected into the bloodstream rather than the brain. This, says Deslys, means that there is an even bigger risk than we thought that vCJD can be passed from one patient to another through contaminated blood transfusions and surgical instruments.

 


 

why do we not want to do TSE transmission studies on chimpanzees $

 

5. A positive result from a chimpanzee challenged severly would likely create alarm in some circles even if the result could not be interpreted for man. I have a view that all these agents could be transmitted provided a large enough dose by appropriate routes was given and the animals kept long enough. Until the mechanisms of the species barrier are more clearly understood it might be best to retain that hypothesis.

 

snip...

 

R. BRADLEY

 


 

1: J Infect Dis 1980 Aug;142(2):205-8

 

Oral transmission of kuru, Creutzfeldt-Jakob disease, and scrapie to nonhuman primates.

 

Gibbs CJ Jr, Amyx HL, Bacote A, Masters CL, Gajdusek DC.

 

Kuru and Creutzfeldt-Jakob disease of humans and scrapie disease of sheep and goats were transmitted to squirrel monkeys (Saimiri sciureus) that were exposed to the infectious agents only by their nonforced consumption of known infectious tissues. The asymptomatic incubation period in the one monkey exposed to the virus of kuru was 36 months; that in the two monkeys exposed to the virus of Creutzfeldt-Jakob disease was 23 and 27 months, respectively; and that in the two monkeys exposed to the virus of scrapie was 25 and 32 months, respectively. Careful physical examination of the buccal cavities of all of the monkeys failed to reveal signs or oral lesions. One additional monkey similarly exposed to kuru has remained asymptomatic during the 39 months that it has been under observation.

 

snip...

 

The successful transmission of kuru, Creutzfeldt-Jakob disease, and scrapie by natural feeding to squirrel monkeys that we have reported provides further grounds for concern that scrapie-infected meat may occasionally give rise in humans to Creutzfeldt-Jakob disease.

 

PMID: 6997404

 


 

Recently the question has again been brought up as to whether scrapie is transmissible to man. This has followed reports that the disease has been transmitted to primates. One particularly lurid speculation (Gajdusek 1977) conjectures that the agents of scrapie, kuru, Creutzfeldt-Jakob disease and transmissible encephalopathy of mink are varieties of a single "virus". The U.S. Department of Agriculture concluded that it could "no longer justify or permit scrapie-blood line and scrapie-exposed sheep and goats to be processed for human or animal food at slaughter or rendering plants" (ARC 84/77)" The problem is emphasised by the finding that some strains of scrapie produce lesions identical to the once which characterise the human dementias"

 

Whether true or not. the hypothesis that these agents might be transmissible to man raises two considerations. First, the safety of laboratory personnel requires prompt attention. Second, action such as the "scorched meat" policy of USDA makes the solution of the acrapie problem urgent if the sheep industry is not to suffer grievously.

 

snip...

 

76/10.12/4.6

 


 

Nature. 1972 Mar 10;236(5341):73-4.

 

Transmission of scrapie to the cynomolgus monkey (Macaca fascicularis).

 

Gibbs CJ Jr, Gajdusek DC.

 

Nature 236, 73 - 74 (10 March 1972); doi:10.1038/236073a0

 

Transmission of Scrapie to the Cynomolgus Monkey (Macaca fascicularis)

 

C. J. GIBBS jun. & D. C. GAJDUSEK

 

National Institute of Neurological Diseases and Stroke, National Institutes of Health, Bethesda, Maryland

 

SCRAPIE has been transmitted to the cynomolgus, or crab-eating, monkey (Macaca fascicularis) with an incubation period of more than 5 yr from the time of intracerebral inoculation of scrapie-infected mouse brain. The animal developed a chronic central nervous system degeneration, with ataxia, tremor and myoclonus with associated severe scrapie-like pathology of intensive astroglial hypertrophy and proliferation, neuronal vacuolation and status spongiosus of grey matter. The strain of scrapie virus used was the eighth passage in Swiss mice (NIH) of a Compton strain of scrapie obtained as ninth intracerebral passage of the agent in goat brain, from Dr R. L. Chandler (ARC, Compton, Berkshire).

 


 


 

Monday, December 1, 2014

 

Germany Bovine Spongiform Encephalopathy BSE CJD TSE Prion disease A Review December 1, 2014

 


 

Friday, November 28, 2014

 

BOVINE SPONGIFORM ENCEPHALOPATHY BSE AKA MAD COW DISEASE PORTUGAL CONFIRMED

 


 

Sunday, October 5, 2014

 

France stops BSE testing for Mad Cow Disease

 


 

Monday, May 5, 2014

 

Brazil BSE Mad Cow disease confirmed OIE 02/05/2014

 


 

Sunday, December 28, 2014

 

*** Reverse Freedom of Information Act request rFOIA FSIS USDA APHIS TSE PRION aka BSE MAD COW TYPE DISEASE December 2014 ***

 


 

Wednesday, December 31, 2014

 

NASDA BSE, CWD, SCRAPIE, TSE, PRION, Policy Statements updated with amendments passed during the NASDA Annual Meeting Updated September 18, 2014

 


 

Sunday, December 28, 2014 CHRONIC WASTING DISEASE CWD TSE PRION DISEASE AKA MAD DEER DISIEASE USDA USAHA INC DECEMBER 28, 2014

 


 

*** HUMAN MAD COW DISEASE nvCJD TEXAS CASE NOT LINKED TO EUROPEAN TRAVEL CDC ***

 

Sunday, November 23, 2014

 

*** Confirmed Variant Creutzfeldt-Jakob Disease (variant CJD) Case in Texas in June 2014 confirmed as USA case NOT European the patient had resided in Kuwait, Russia and Lebanon. The completed investigation did not support the patient's having had extended travel to European countries, including the United Kingdom, or travel to Saudi Arabia. The specific overseas country where this patient’s infection occurred is less clear largely because the investigation did not definitely link him to a country where other known vCJD cases likely had been infected.

 


 

Sunday, December 14, 2014

 

ALERT new variant Creutzfeldt Jakob Disease nvCJD or vCJD, sporadic CJD strains, TSE prion aka Mad Cow Disease United States of America Update December 14, 2014 Report

 


 

Sunday, May 10, 2009

 

Meeting of the Transmissible Spongiform Encephalopathies Committee On June 12, 2009 (Singeltary submission)

 

TO : william.freas@fda.hhs.gov

 


 

Harvard Risk Assessment of Bovine Spongiform Encephalopathy Update, October 31, 2005 INTRODUCTION The United States Department of Agriculture’s Food Safety and Inspection Service (FSIS) held a public meeting on July 25, 2006 in Washington, D.C. to present findings from the Harvard Risk Assessment of Bovine Spongiform Encephalopathy Update, October 31, 2005 (report and model located on the FSIS website:

 


 

Comments on technical aspects of the risk assessment were then submitted to FSIS. Comments were received from Food and Water Watch, Food Animal Concerns Trust (FACT), Farm Sanctuary, R-CALF USA, Linda A Detwiler, and Terry S. Singeltary. This document provides itemized replies to the public comments received on the 2005 updated Harvard BSE risk assessment. Please bear the following points in mind:

 


 

Tuesday, December 30, 2014

 

TSEAC USA Reason For Recalls Blood products, collected from a donors considered to be at increased risk for Creutzfeldt-Jakob Disease (CJD), were distributed END OF YEAR REPORT 2014

 


 

From: Terry S. Singeltary Sr.

 

To: FREAS@CBER.FDA.GOV

 

Cc: william.freas@fda.hhs.gov ; rosanna.harvey@fda.hhs.gov

 

Sent: Friday, December 01, 2006 2:59 PM

 

Subject: Re: TSE advisory committee for the meeting December 15, 2006 [TSS SUBMISSION

 

snip...

 

ONE FINAL COMMENT PLEASE, (i know this is long Dr. Freas but please bear with me)

 

THE USA is in a most unique situation, one of unknown circumstances with human and animal TSE. THE USA has the most documented TSE in different species to date, with substrains growing in those species (BSE/BASE in cattle and CWD in deer and elk, there is evidence here with different strains), and we know that sheep scrapie has over 20 strains of the typical scrapie with atypical scrapie documented and also BSE is very likely to have passed to sheep. all of which have been rendered and fed back to animals for human and animal consumption, a frightening scenario. WE do not know the outcome, and to play with human life around the globe with the very likely TSE tainted blood from the USA, in my opinion is like playing Russian roulette, of long duration, with potential long and enduring consequences, of which once done, cannot be undone.

 

These are the facts as i have come to know through daily and extensive research of TSE over 9 years, since 12/14/97. I do not pretend to have all the answers, but i do know to continue to believe in the ukbsenvcjd only theory of transmission to humans of only this one strain from only this one TSE from only this one part of the globe, will only lead to further failures, and needless exposure to humans from all strains of TSE, and possibly many more needless deaths from TSE via a multitude of proven routes and sources via many studies with primates and rodents and other species. ...

 

Terry S. Singeltary Sr. P.O. Box 42 Bacliff, Texas USA 77518

 

snip... 48 pages...

 


 

Wednesday, October 17, 2007

 

TSEAC MEETINGS

 

----- Original Message -----

 

From: Terry S. Singeltary Sr.

 

To: FREAS@CBER.FDA.GOV

 

Cc: william.freas@fda.hhs.gov ; rosanna.harvey@fda.hhs.gov

 

Sent: Wednesday, November 29, 2006 1:24 PM

 

Subject: TSE advisory committee for the meeting December 15, 2006 [TSSSUBMISSION]November 29, 2006

 

Greetings FDA, DHH, Dr. Freas, and Dr. Harvey et al,

 

a kind and warm Holiday Greetings to you all.i kindly wish to submit the following to the TSE advisory committee for the meeting December 15, 2006, about the assessment for potential exposure to vCJD in human plasma-derivedantihemophilic factor (FVIII) productsmanufactured from U.S. plasma donors and related communication material ;

 


 

i see the media picked up on this as a 'low risk', from what the gov. agency perceived to be to them;

 


 

however, i seem to disagree. from my primitive ciphering, i see it another way. this is a huge catastrophic risk. 3 in 160 is 1.9%. so call that 2% which is 1 in 50 or twenty per thousand or 20,000 per million. also, wha tabout the mixed genotypes/mixed susceptibility?

 

what about the silent carriers that donated tainted blood?

 

what about the sporadic CJDs of UNKNOWN strain or phenotype?

 

this risk assessment is just more BSe to me. just another in a long line of industry fed crap. i pray that my assessment is the one that is wrong. but it is THEY who roll the dice with your life. it is THEY who refuse to regulate an industry that has run amok. just from are call aspect of potentially tainted blood, and these are just recent recalls ;

 

PRODUCT

 

Source Plasma, Recall # B-0054-7CODEUnits: 03MMNC5465, 03MMNC6361, 03MMNC6801, 03MMNC7510, 03MMNC7891,03MMNC8252, 03MMNC8801, 03MMNC9144, 03MMND1122, 03MMND1478, 03MMND1969,03MMND2350, 03MMND2825, 03MMND3211, 03MMND3708, 03MMND4072, 03MMND4588,03MMND4831, 03MMND5320, 03MMND5719, 03MMND6268, 03MMND6683, 03MMND7228,03MMND7656, 03MMND8211, 03MMND8652, 03MMND9195, 03MMND9618, 03MMNE0628,03MMNE0884, 03MMNE1597, 03MMNE1979, 03MMNE2644, 03MMNE3064, 03MMNE3707,03MMNE4122, 03MMNE4750, 03MMNE5080, 03MMNE5876, 03MMNE6218, 03MMNE7189,03MMNE7587, 03MMNE8027, 03MMNE8645, 03MMNE9029, 03MMNE9641, 03MMNE9979,03MMNF0491, 03MMNF0685, 03MMNF0937, 03MMNF1260, 04MMNA0351, 04MMNA0707,04MMNA1241, 04MMNA1650, 04MMNA2291, 04MMNA2646, 04MMNA3340, 04MMNA3719,04MMNA4312, 04MMNA4683, 04MMNA5298, 04MMNA5750, 04MMNA6407, 04MMNA6816,04MMNA7482, 04MMNA7915, 04MMNA8632, 04MMNA9076, 04MMNA9723, 04MMNB0063,04MMNB0696, 04MMNB1100, 04MMNB1845, 04MMNB2285, 04MMNB3035, 04MMNB3485,04MMNB4213, 04MMNB4672, 04MMNB5841, 04MMNB6652, 04MMNB7162, 04MMNB7930,04MMNB8453, 04MMNB9239, 04MMNB9747, 04MMNC0456, 04MMNC0931, 04MMNC1578

 

RECALLING FIRM/MANUFACTURER

 

BioLife Plasma Services, L.P., Mankato, MN, by facsimile on June 4, 2004.

 

Firm initiated recall is complete.

 

REASON

 

Blood products, collected from a donor who was at increased risk for new variant Creutzfeldt-Jakob Disease (nvCJD), were distributed.

 

VOLUME OF PRODUCT IN COMMERCE

 

89 units

 

DISTRIBUTION

 

CA and Austria

 

END OF ENFORCEMENT REPORT FOR October 25, 2006

 

###

 


 

SNIP...

 

Greetings again Dr. Freas et al at FDA,

 

WITH new atypical TSE in the bovine, in the sheep, goat, and humans, and the fact that the new BASE TSE in cattle being very very similar to sporadic CJD, rather than the nvCJD, the fact that now science showing the TSE agent of the atypical cattle in Japan showing infectivity other than CNS tissue, the fact that the latest Texas mad cow and the recent Alabama mad cow both being of the atypical strain, it would seem prudent to include all human TSE in the blood ban, in my opinion. with sporadic CJD, you have many strains and or phenotypes, some of which are 'UNKNOWN', so we do not know how this will transmit, what tissues are infectious and or if blood transmits. that's the bottomline, however it has been reported that the BASE is more virulent to humans.With this, and the fact that sporadic CJD has tripled in the past few years or so, i see itas being prudent to take serious and immediate action ;

 


 

2001 Singeltary Submission to FDA on blood risk factors from TSE prion aka mad cow type disease

 

PDF]Freas, William TSS SUBMISSION

 

File Format: PDF/Adobe Acrobat -

 

Page 1. J Freas, William From: Sent: To: Subject: Terry S. Singeltary

 

Sr. [flounder@wt.net] Monday, January 08,200l 3:03 PM freas ...

 


 

Tuesday, February 8, 2011

 

U.S.A. 50 STATE BSE MAD COW CONFERENCE CALL Jan. 9, 2001

 


 

Thursday, February 24, 2011

 

The risk of variant Creutzfeldt-Jakob disease among UK patients with bleeding disorders, known to have received potentially contaminated plasma products

 


 

BSE INQUIRY DFAs

 


 

Sunday, May 18, 2008

 

BSE Inquiry DRAFT FACTUAL ACCOUNT DFA

 

BSE Inquiry DRAFT FACTUAL ACCOUNTS DFA's

 


 

Sunday, May 18, 2008

 

***BSE, CJD, and Baby foods (the great debate 1999 to 2005)

 


 

Sunday, May 18, 2008

 

***MAD COW DISEASE BSE CJD CHILDREN VACCINES

 


 

Sadly, HOW ALL THIS MAD COW MADNESS GOT STARTED, was importing and exporting these TSE mad cow type prions all over the globe. NOW, thanks to the OIE and the USDA inc. et al, they just made it all legal now $$$

 

UK EXPORTS OF MBM TO WORLD Bovine Spongiform Encephalopathy BSE TSE Prion aka Mad Cow Disease

 

Bovine Spongiform Encephalopathy BSE TSE Prion aka Mad Cow Disease

 

Subject: UK EXPORTS OF MBM TO WORLD

 

UK EXPORTS OF MBM TO WORLD

 


 


 


 

OTHERS

 

BEEF AND VEAL

 


 


 


 

LIVE CATTLE

 


 

FATS

 


 

EMBRYOS

 


 

GELATIN ETC

 


 

SEMEN

 


 

MEAT

 


 

when sound science was bought off by junk science, in regards to the BSE TSE prion mad cow type disease, by the USDA, CFIA, WHO, OIE, et al. $$$

 

when the infamous, and fraudulently USDA, FSIS, APHIS, FDA, gold card was taken away that infamous day in December of 2003, all cards were off the table, it was time to change the science, and change they did. ...tss

 

snip. ...please see full text ;

 

Thursday, June 6, 2013

 

BSE TSE PRION USDA FDA MAD COW FEED COMPLIANCE REPORT and NAI, OAI, and VAI ratings as at June 5, 2013

 


 

Saturday, August 30, 2014

 

Maine Firm Recalls Ribeye and Carcass Products That May Contain Specified Risk Materials SRM TSE PRION aka mad cow type disease

 


 

Friday, December 19, 2014

 

Rancho Alleged Cancerous Eyeball Case Going To Trial

 


 

Thursday, November 28, 2013

 

Department of Justice Former Suppliers of Beef to National School Lunch Program Settle Allegations of Improper Practices and Mistreating Cows

 


 

seems USDA NSLP et al thought that it would be alright, to feed our children all across the USA, via the NSLP, DEAD STOCK DOWNER COWS, the most high risk cattle for mad cow type disease, and other dangerous pathogens, and they did this for 4 years, that was documented, then hid what they did by having a recall, one of the largest recalls ever, and they made this recall and masked the reason for the recall due to animal abuse (I do not condone animal abuse), not for the reason of the potential for these animals to have mad cow BSE type disease (or other dangerous and deadly pathogens). these TSE prion disease can lay dormant for 5, 10, 20 years, or longer, WHO WILL WATCH OUR CHILDREN FOR THE NEXT 5 DECADES FOR CJD ???

 

Saturday, September 21, 2013

 

Westland/Hallmark: 2008 Beef Recall A Case Study by The Food Industry Center January 2010 THE FLIM-FLAM REPORT

 


 

DID YOUR CHILD CONSUME SOME OF THESE DEAD STOCK DOWNER COWS, THE MOST HIGH RISK FOR MAD COW DISEASE ??? this recall was not for the welfare of the animals. ...tss you can check and see here ; (link now dead, does not work...tss)

 


 

try this link ;

 


 

Sunday, November 13, 2011

 

*** California BSE mad cow beef recall, QFC, CJD, and dead stock downer livestock

 


 

Thursday, February 13, 2014

 

HSUS VS USDA ET AL BAN DOWNER CALVES FOR HUMAN CONSUMPTION (*veal) and potential BSE risk factor there from

 


 

Saturday, November 10, 2012

 

Wisconsin Firm Recalls Beef Tongues That May Contain Specified Risk Materials Nov 9, 2012 WI Firm Recalls Beef Tongues

 


 

Saturday, July 23, 2011

 

CATTLE HEADS WITH TONSILS, BEEF TONGUES, SPINAL CORD, SPECIFIED RISK MATERIALS (SRM's) AND PRIONS, AKA MAD COW DISEASE

 


 

Sunday, October 18, 2009

 

Wisconsin Firm Recalls Beef Tongues That Contain Prohibited Materials SRM WASHINGTON, October 17, 2009

 


 

Thursday, October 15, 2009

 

Nebraska Firm Recalls Beef Tongues That Contain Prohibited Materials SRM WASHINGTON, Oct 15, 2009

 


 

Thursday, June 26, 2008

 

Texas Firm Recalls Cattle Heads That Contain Prohibited Materials

 


 

Tuesday, July 1, 2008

 

Missouri Firm Recalls Cattle Heads That Contain Prohibited Materials SRMs

 


 

Friday, August 8, 2008

 

Texas Firm Recalls Cattle Heads That Contain Prohibited Materials SRMs 941,271 pounds with tonsils not completely removed

 


 

Saturday, April 5, 2008

 

SRM MAD COW RECALL 406 THOUSAND POUNDS CATTLE HEADS WITH TONSILS KANSAS

 


 

Wednesday, April 30, 2008

 

Consumption of beef tongue: Human BSE risk associated with exposure to lymphoid tissue in bovine tongue in consideration of new research findings

 


 

Wednesday, April 30, 2008

 

Consumption of beef tongue: Human BSE risk associated with exposure to lymphoid tissue in bovine tongue in consideration of new research findings

 


 

Friday, October 15, 2010

 

BSE infectivity in the absence of detectable PrPSc accumulation in the tongue and nasal mucosa of terminally diseased cattle

 


 

SPECIFIED RISK MATERIALS SRMs

 


 

Thursday, May 30, 2013

 

World Organization for Animal Health (OIE) has upgraded the United States' risk classification for mad cow disease to "negligible" from "controlled", and risk further exposing the globe to the TSE prion mad cow type disease

 

U.S. gets top mad-cow rating from international group and risk further exposing the globe to the TSE prion mad cow type disease

 


 


 

Tuesday, July 2, 2013

 

APHIS USDA Administrator Message to Stakeholders: Agency Vision and Goals Eliminating ALL remaining BSE barriers to export market

 


 

Monday, June 18, 2012

 

R-CALF Submits Incomplete Comments Under Protest in Bizarre Rulemaking “Bovine Spongiform Encephalopathy; Importation of Bovines and Bovine Products”

 


 

Tuesday, July 17, 2012

 

O.I.E. BSE, CWD, SCRAPIE, TSE PRION DISEASE Final Report of the 80th General Session, 20 - 25 May 2012

 


 

Thursday, December 20, 2012

 

OIE GROUP RECOMMENDS THAT SCRAPE PRION DISEASE BE DELISTED AND SAME OLD BSe WITH BOVINE MAD COW DISEASE

 


 

Monday, November 30, 2009

 

*** USDA AND OIE COLLABORATE TO EXCLUDE ATYPICAL SCRAPIE NOR-98 ANIMAL HEALTH CODE, DOES NOT SURPRISE ME $

 


 

Saturday, July 6, 2013

 

*** Small Ruminant Nor98 Prions Share Biochemical Features with Human Gerstmann-Sträussler-Scheinker Disease and Variably Protease-Sensitive Prionopathy

 

Research Article

 


 

Sunday, December 7, 2014

 

Scientific update on the potential for transmissibility of non-prion protein misfolding diseases PRIONOIDS

 


 

Self-Propagative Replication of Ab Oligomers Suggests Potential Transmissibility in Alzheimer Disease

 

Received July 24, 2014; Accepted September 16, 2014; Published November 3, 2014

 

Singeltary comment ;

 


 

Saturday, December 13, 2014

 

Terry S. Singeltary Sr. Publications TSE prion disease

 

Diagnosis and Reporting of Creutzfeldt-Jakob Disease

 

Singeltary, Sr et al. JAMA.2001; 285: 733-734. Vol. 285 No. 6, February 14, 2001 JAMA

 

snip...

 


 

Terry S. Singeltary Sr. Texas USA 77518 flounder9@verizon.net

Thursday, December 25, 2014

Bovine spongiform encephalopathy, Romania Confirmed

Re: [BSE-L] ROU 20-06-14 OIE Alert - Alerta - Alerte - Bovine spongiform encephalopathy - Encéphalopathie spongiforme bovine - Encefalopatía espongiforme bovina (atypical BSE L-type)
 
 
UPDATE December 25, 2014
 
Bovine spongiform encephalopathy, Romania
 
Information received on 23/12/2014 from Dr Maximilian Dragan, General Director, National Sanitary Veterinary and Food Safety Authority, Ministry of Agriculture and Food, Bucharest, Romania
Summary
Report type Follow-up report No. 3
Date of start of the event 06/05/2014
Date of pre-confirmation of the event 09/06/2014
Report date 23/12/2014
Date submitted to OIE 23/12/2014
Reason for notification First occurrence of a listed disease
Manifestation of disease Sub-clinical infection
Causal agent Prion
Nature of diagnosis Laboratory (advanced)
This event pertains to the whole country
Related reports Immediate notification (20/06/2014) Follow-up report No. 1 (20/10/2014) Follow-up report No. 2 (27/10/2014) Follow-up report No. 3 (23/12/2014)
New outbreaks (1)
Outbreak 1 Reci, COVASNA
Date of start of the outbreak 22/12/2014
Outbreak status Continuing (or date resolved not provided)
Epidemiological unit Backyard
Affected animals
Species Susceptible Cases Deaths Destroyed Slaughtered
Cattle 1 0 1 0
Affected population One bovine slaughtered at SC Neval Pietrosita, Dambovita county. No clinical signs.
Summary of outbreaks Total outbreaks: 1
Total animals affected
Species Susceptible Cases Deaths Destroyed Slaughtered
Cattle 1 0 1 0
Outbreak statistics
Species Apparent morbidity rate Apparent mortality rate Apparent case fatality rate Proportion susceptible animals lost*
Cattle ** ** 0.00% **
*Removed from the susceptible population through death, destruction and/or slaughter
**Not calculated because of missing information
Epidemiology
Source of the outbreak(s) or origin of infection
  • Unknown or inconclusive
Epidemiological comments One bovine without clinical signs with identification number RO155000108204 slaughtered in a slaughter house in Dambovita County. Follow up to identify the cohort will be done. Control measures in accordance with the EU regulation (CE) 999/2001.
Control measures
Measures applied
  • Screening
  • No vaccination
  • No treatment of affected animals
Measures to be applied
  • No other measures
Diagnostic test results
Laboratory name and type Species Test Test date Result
Institute for Diagnostic and Animal Health (National laboratory) Cattle rapid tests 22/12/2014 Positive
Institute for Diagnostic and Animal Health (National laboratory) Cattle western blot 23/12/2014 Positive
Future Reporting
The event is continuing. Weekly follow-up reports will be submitted.
Map of outbreak locations
http://www.oie.int/wahis_2/public/wahid.php/Reviewreport/Review?page_refer=MapEventSummary&reportid=16819
 
 
Tuesday, December 23, 2014
 
FDA PART 589 -- SUBSTANCES PROHIBITED FROM USE IN ANIMAL FOOD OR FEED VIOLATIONS OFFICIAL ACTION INDICATED OAI UPDATE DECEMBER 2014 BSE TSE PRION
 
 
Wednesday, December 24, 2014
 
National Scrapie Eradication Program November 2014 Monthly Report Fiscal Year 2015
 
 
Tuesday, December 16, 2014
 
Evidence for zoonotic potential of ovine scrapie prions
 
Scrapie from sheep could infect humans with 'mad cow disease', study finds
 
 
 
 
*** HUMAN MAD COW DISEASE nvCJD TEXAS CASE NOT LINKED TO EUROPEAN TRAVEL CDC ***
 
Sunday, November 23, 2014
 
*** Confirmed Variant Creutzfeldt-Jakob Disease (variant CJD) Case in Texas in June 2014 confirmed as USA case NOT European
 
the patient had resided in Kuwait, Russia and Lebanon. The completed investigation did not support the patient's having had extended travel to European countries, including the United Kingdom, or travel to Saudi Arabia. The specific overseas country where this patient’s infection occurred is less clear largely because the investigation did not definitely link him to a country where other known vCJD cases likely had been infected.
 
 
Sunday, December 14, 2014
 
ALERT new variant Creutzfeldt Jakob Disease nvCJD or vCJD, sporadic CJD strains, TSE prion aka Mad Cow Disease United States of America Update December 14, 2014 Report
 
 
Saturday, December 13, 2014
 
Terry S. Singeltary Sr. Publications TSE prion disease
 
for my files...tss
 
Diagnosis and Reporting of Creutzfeldt-Jakob Disease
 
Singeltary, Sr et al. JAMA.2001; 285: 733-734. Vol. 285 No. 6, February 14, 2001 JAMA
 
snip...
 
 
 
TSS
 
 
Sent: Saturday, June 21, 2014 11:39 AM
Subject: [BSE-L] ROU 20-06-14 OIE Alert - Alerta - Alerte - Bovine spongiform encephalopathy - Encéphalopathie spongiforme bovine - Encefalopatía espongiforme bovina (atypical BSE L-type)
 
 
Sent: Friday, June 20, 2014 11:10 AM
 
 
Subject: ROU 20-06-14 OIE Alert - Alerta - Alerte - Bovine spongiform encephalopathy - Encéphalopathie spongiforme bovine - Encefalopatía espongiforme bovina
 
English PDF reports Français Rapports PDF Español informes PDF
 
Bovine spongiform encephalopathy ,Romania Information received on 20/06/2014 from Dr Cristian Duicu, Director General, National Veterinary and Food Safety Authority for General Sanitary Veterinary Department, Ministry of Agriculture and Food, Bucharest , Romania
 
Summary Report type Immediate notification Date of start of the event 06/05/2014 Date of pre-confirmation of the event 09/06/2014 Report date 20/06/2014 Date submitted to OIE 20/06/2014 Reason for notification First occurrence of a listed disease Manifestation of disease Sub-clinical infection Causal agent Prion (atypical BSE L-type) Nature of diagnosis Laboratory (advanced) This event pertains to the whole country
 
New outbreaks Summary of outbreaks Total outbreaks: 1 Outbreak Location CLUJ ( Deusu, Deusu, Chinteni ) Total animals affected Species Susceptible Cases Deaths Destroyed Slaughtered Cattle 1 0 0 0 Outbreak statistics Species Apparent morbidity rate Apparent mortality rate Apparent case fatality rate Proportion susceptible animals lost* Cattle ** ** 0.00% **
 
* Removed from the susceptible population through death, destruction and/or slaughter; ** Not calculated because of missing information;
 
Epidemiology Source of the outbreak(s) or origin of infection Unknown or inconclusive Epidemiological comments The investigation is ongoing.
 
Control measures Measures applied Movement control inside the country No vaccination No treatment of affected animals Measures to be applied No other measures
 
Diagnostic test results Laboratory name and type Institute for Diagnosis and Animal Health ( National laboratory ) Tests and results Species Test Test date Result Cattle western blot 09/06/2014 Positive Laboratory name and type Animal Health and Veterinary Laboratories Agency (AHVLA), Weybridge, United Kingdom ( OIE’s Reference Laboratory ) Tests and results Species Test Test date Result Cattle immunohistochemical test 12/06/2014 Positive Cattle western blot 12/06/2014 Positive
 
Future Reporting The event is continuing. Weekly follow-up reports will be submitted.
 
 
Encéphalopathie spongiforme bovine ,Roumanie Information reçue le 20/06/2014 de Dr Cristian Duicu, Director General, National Veterinary and Food Safety Authority for General Sanitary Veterinary Department, Ministry of Agriculture and Food, Bucharest , Roumanie
 
Résumé Type de rapport Notification immédiate Date de début de l’événement 06/05/2014 Date de pré-confirmation de l´événement 09/06/2014 Date du rapport 20/06/2014 Date d'envoi à l'OIE 20/06/2014 Raison de notification Apparition pour la première fois d’une maladie appartenant à la liste de l'OIE Manifestation de la maladie Infection sub-clinique Agent causal Prion (ESB atypique type L) Nature du diagnostic Tests approfondis en laboratoire (i.e. virologie, microscopie électronique, biologie moléculaire, immunologie) Cet événement se rapporte à tout le pays
 
Nouveaux foyers Récapitulatif des foyers Nombre total de foyers : 1 Localisation du foyer CLUJ ( Deusu, Deusu, Chinteni ) Nombre total d'animaux atteints Espèce(s) Sensibles Cas Morts Détruits Abattus Bovins 1 0 0 0 Statistiques sur le foyer Espèce(s) Taux de morbidité apparent Taux de mortalité apparent Taux de fatalité apparent Proportion d'animaux sensibles perdus* Bovins ** ** 0.00% **
 
* Soustraits de la population sensible suite à la mort, à l´abattage et/ou à la destruction; ** Non calculé par manque de données;
 
Epidémiologie Source du/des foyer(s) ou origine de l´infection Inconnue ou incertaine Autres renseignements épidémiologiques / Commentaires L'enquête est en cours.
 
Mesures de lutte Mesure de lutte appliquées Restriction des déplacements à l'intérieur du pays Pas de vaccination Aucun traitement des animaux atteints Mesures à appliquer Aucune autre mesure
 
Résultats des tests de diagnostics Nom du laboratoire et type Agence des Laboratoires vétérinaires et santé animale (AHVLA), Weybridge (Royaume-Uni) ( Laboratoire de référence de l’OIE ) Tests et résultats Espèce(s) Test Date du test Résultat Bovins examen immunohistochimique 12/06/2014 Positif Bovins western blot 12/06/2014 Positif Nom du laboratoire et type Institut de diagnostic et de santé animale ( Laboratoire national ) Tests et résultats Espèce(s) Test Date du test Résultat Bovins western blot 09/06/2014 Positif
 
Rapports futurs Cet événement se poursuit. Des rapports de suivi hebdomadaires devront être envoyés.
 
 
Encefalopatía espongiforme bovina ,Rumania Información recibida el 20/06/2014 desde Dr Cristian Duicu, Director General, National Veterinary and Food Safety Authority for General Sanitary Veterinary Department, Ministry of Agriculture and Food, Bucharest , Rumania
 
Resumen Tipo de informe Notificación inmediata Fecha del inicio del evento 06/05/2014 Fecha de pre-confirmación del evento 09/06/2014 Fecha del informe 20/06/2014 Fecha de envio del informe a la OIE 20/06/2014 Motivo de la notificación Aparición por primera vez de una enfermedad de la Lista de la OIE Manifestación de la enfermedad Infección sub-clínica Agente causal Prion (EEB atípica tipo L) Naturaleza del diagnóstico Pruebas de diagnóstico de laboratorio avanzadas (ej. virología, microscopía electrónica, biología molecular e inmunología) Este evento concierne todo el país
 
Nuevos focos Resumen de los focos Número total de focos: 1 Localización del foco CLUJ ( Deusu, Deusu, Chinteni ) Número total de animales afectados Especies Susceptibles Casos Muertos Destruidos Sacrificados Bovinos 1 0 0 0 Estadística del foco Especies Tasa de morbilidad aparente Tasa de mortalidad aparente Tasa de fatalidad aparente Proporción de animales susceptibles perdidos* Bovinos ** ** 0.00% **
 
* Descontados de la población susceptible a raíz de su muerte, destrucción o sacrificio; ** No calculado por falta de datos;
 
Epidemiología Fuente del o de los focos u origen de la infección Desconocida o no concluyente Otros detalles epidemiológicos / comentarios Se está investigando.
 
Medidas de Control Medidas implementadas Restricción de los movimientos en el interior del país Vacunación: no Ningún tratamiento de los animales afectados Medidas para implementar Ninguna otra medida
 
Resultados de las pruebas diagnósticas Nombre y tipo de laboratorio Agencia de laboratorios veterinarios y sanidad animal (AHVLA), Weybridge (Reino Unido) ( Laboratorio de referencia de la OIE ) Pruebas y resultados Especies Prueba Fecha de la prueba Resultados Bovinos examen inmunohistoquímico 12/06/2014 Positivo Bovinos western blot 12/06/2014 Positivo Nombre y tipo de laboratorio Instituto de diagnóstico y sanidad animal ( Laboratorio nacional ) Pruebas y resultados Especies Prueba Fecha de la prueba Resultados Bovinos western blot 09/06/2014 Positivo
 
Informes futuros El episodio continúa. Informes de seguimiento semanales serán enviados
 
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Press releases
 
Brussels, 16 May 2001
 
BSE: Scientists publish risk assessments for Costa Rica, Kenya, Slovenia and Romania
 
The Scientific Steering Committee (SSC) advising the European Commission on BSE related issues has today published its opinion on the Geographical Risk of Bovine Spongiform Encephalopathy (GBR) in Costa Rica, Kenya, Slovenia and Romania. The evaluation of the geographical risk of presence of BSE focuses on the risk for animals to incubate the disease. The Committee concludes that is highly unlikely that cattle infected with the BSE agent are present in domestic herds of Costa Rica (GBR level I). They found that this is unlikely but not excluded in the herds of Kenya and Slovenia (GBR level II) and that it is likely that BSE is present in the cattle herds of Romania (GBR level III) although this is not yet confirmed. Slovenia is the first accession country that is classified as GBR level II. All other accession countries evaluated so far have been classified at level III of Geographical BSE Risk. Similarly, all EU Member States are classified at level III except for Sweden, Finland and Austria (level II) and United Kingdom and Portugal (level IV).
 
The Committee found that Slovenia has since 1992 imported 2.400 live cattle notably from Germany, and imported small amounts of MBM. The Slovenian authorities have been able to trace most of these cattle imports and to demonstrate that many of them are still alive. They also showed that reasonably effective controls on the rendering of MBM were in place at least as of 1996, and probably also before that date. In addition, a first feed ban to ruminants was introduced in 1996. It is therefore regarded unlikely but not excluded that the BSE agent could have been recycled, but not amplified, in Slovenia between 1992 and January 2001, when a complete feed ban was put in place. Romania has imported higher numbers of live cattle (about 22,000 tons) and meat-and-bone-meal (about 10,000 tons) from EU countries where the presence of BSE has since been confirmed. Although risk management measures were taken as of 1996, their effective enforcement has not been demonstrated. Therefore it is regarded likely that Romanian cattle herds were exposed to potentially BSE contaminated feed and subsequently infected.
 
Kenya has received meat and bone meal exports notably between 1987-1990 from the UK and since 1994 from Belgium, Denmark and the Netherlands. The data made available to the SSC do not exclude that some of this MBM has reached domestic cattle. The conclusion of the assessment for Costa Rica is based on data demonstrating that BSE infectivity is highly unlikely to have reached the country and hence the domestic cattle population. Only minor quantities of potentially infected live cattle (35 from Spain) or potentially contaminated meat-and-bone meal (5 tonnes) were imported into the country.
 
The SSC recommends that BSE related aspects are included in the programme of future inspection missions of the Food and Veterinary Office, as far as feasible, to obtain confirmation of the information received from the national authorities in the countries concerned. For the time being, the scientists underline, their assessment has to be based on the information provided by the assessed countries. As far as possible all data have been evaluated and verified in close co-operation with the countries concerned, and checked against other sources in an open and transparent manner. Data on imports provided by the countries under evaluation have for example been compared with export data as recorded by EUROSTAT, the EU Statistical Office, and with export data provided by the UK authorities.
 
The evaluation of the GBR in these third countries was made on the basis of the same method and assessment process as described by the SSC in its July 2000 opinion on the GBR( 1 ). In the July-opinion the scientists already assessed the GBR risk in all EU Member States except Greece, and a first series of third countries( 2 ). An assessment for Uruguay was published in January; assessments for Botswana, Lithuania, Namibia, Nicaragua, and Swaziland in February, and for Albania, Brazil, Colombia, Republic of Cyprus, Czech Republic, Estonia, Hungary, India, Mauritius, Pakistan, Poland, Singapore and Slovakia in April this year.
 
The full text of the opinions is available at:
 
 
Released on 29/05/2001
 
Updated Overview of third countries according to Geographical BSE risk classification
 
Category I: Highly unlikely to present a BSE risk
 
Argentina Australia Botswana Brazil Chile Costa Rica Namibia Nicaragua Norway New Zealand Paraguay Singapore Swaziland Uruguay
 
Category II: Unlikely, but a BSE risk cannot be excluded
 
Canada Colombia India Kenya Mauritius Pakistan Slovenia USA
 
Category III: likely to present a BSE risk, even if not confirmed, or presenting a low level of confirmed BSE risk
 
Albania Cyprus Czech Republic Estonia Hungary Lithuania Poland Romania Slovak Republic Switzerland
 
Category IV: BSE risk confirmed at a high level
 
None
 
----------------------------------------
 
1 see IP of August1, 2000 at :
 
 
2 Argentina, Australia, Canada, Chile, Norway, New Zealand, Paraguay, Switzerland, USA
 
 
2004 BSE GBR
 
 
 
 
Opinion of the Scientific Steering Committee on the GEOGRAPHICAL RISK OF BOVINE SPONGIFORM ENCEPHALOPATHY (GBR) in Romania Adopted on 11/05/2001
 
 
 
FINAL REPORT OF AN AUDIT CARRIED OUT IN ROMANIA FROM 07 TO 18 FEBRUARY 2011 IN ORDER TO EVALUATE MEASURES CONCERNING BOVINE SPONGIFORM ENCEPHALOPATHY (BSE)
 
Executive Summary This report describes the outcome of an audit carried out by the Food and Veterinary Office (FVO) in Romania, from 7 to 18 February 2011.
 
The objective of the audit was to evaluate the implementation of requirements concerning Bovine Spongiform Encephalopathy (BSE), as laid down in Regulation (EC) No 999/2001.
 
In terms of scope, the audit concentrated on BSE epidemio-surveillance in bovines, measures taken after suspicion/confirmation of BSE, removal and handling of specified risk material (SRM) from bovines, and the prohibition of feeding products of animal origin to farmed animals and exceptions applicable to this ban. The evaluation included measures taken in response to the recommendations made in a previous FVO audit regarding the afore-mentioned issues.
 
Overall, the report concludes that very limited progress has been made in order to address the recommendations of the previous FVO audit. In particular, BSE active epidemio-surveillance and compliance with SRM rules are significantly affected by the lack of arrangements for the collection of brain samples and SRM at backyard farms, where the majority of the bovine population is kept. There are also weaknesses concerning feed-ban controls.
 
The report makes a number of recommendations addressed to the Romanian competent authorities, aimed at rectifying the shortcomings identified and further enhancing the implementing and control measures in place.
 
 
 
 
 
SEE ;
 
Friday, January 17, 2014
 
Annual report of the Scientific Network on BSE-TSE EFSA, Question No EFSA-Q-2013-01004, approved on 11 December 2013 TECHNICAL REPORT
 
 
 
TSS

Saturday, December 6, 2014

Detection of Bovine Central Nervous System Tissues in Rendered Animal By-Products by One-Step Real-Time Reverse Transcription PCR Assay

Detection of Bovine Central Nervous System Tissues in Rendered Animal By-Products by One-Step Real-Time Reverse Transcription PCR Assay

 

Authors: Andrievskaia, Olga1; Tangorra, Erin2

 

Source: Journal of Food Protection®, Number 12, December 2014, pp. 2012-2218, pp. 2088-2097(10)

 

Publisher: International Association for Food Protection

 

Abstract:

 

Contamination of rendered animal byproducts with central nervous system tissues (CNST) from animals with bovine spongiform encephalopathy is considered one of the vehicles of disease transmission. Removal from the animal feed chain of CNST originated from cattle of a specified age category, species-labeling of rendered meat products, and testing of rendered products for bovine CNST are tasks associated with the epidemiological control of bovine spongiform encephalopathy. A single-step TaqMan real-time reverse transcriptase (RRT) PCR assay was developed and evaluated for specific detection of bovine glial fibrillary acidic protein (GFAP) mRNA, a biomarker of bovine CNST, in rendered animal by-products. An internal amplification control, mammalian b -actin mRNA, was coamplified in the duplex RRT-PCR assay to monitor amplification efficiency, normalize amplification signals, and avoid false-negative results. The functionality of the GFAP mRNA RRT-PCR was assessed through analysis of laboratory-generated binary mixtures of bovine central nervous system (CNS) and muscle tissues treated under various thermal settings imitating industrial conditions. The assay was able to detect as low as 0.05 % (wt/wt) bovine brain tissue in binary mixtures heat treated at 110 to 130°C for 20 to 60 min. Further evaluation of the GFAP mRNA RRT-PCR assay involved samples of industrial rendered products of various species origin and composition obtained from commercial sources and rendering plants. Low amounts of bovine GFAP mRNA were detected in several bovine-rendered products, which was in agreement with declared species composition. An accurate estimation of CNS tissue content in industrial-rendered products was complicated due to a wide range of temperature and time settings in rendering protocols. Nevertheless, the GFAP mRNA RRT-PCR assay may be considered for bovine CNS tissue detection in rendered products in combination with other available tools (for example, animal age verification) in inspection programs.

 

 Document Type: Research Article

 


 

Affiliations: 1: Canadian Food Inspection Agency, Ottawa Laboratory (Fallowfield), 3851 Fallowfield Road, Ottawa, Ontario, Canada K2H 8P9;, Email: Olga.Andrievskaia@inspection.gc.ca 2: Canadian Food Inspection Agency, Ottawa Laboratory (Fallowfield), 3851 Fallowfield Road, Ottawa, Ontario, Canada K2H 8P9

 

Publication date: December 1, 2014

 


 

Friday, May 18, 2012

 

December 23, 2002 at 12:12 pm PST

 

Re: USA ruminant-to-ruminant feed ban warning letters ???

 

December 23, 2002 at 12:12 pm PST

 

Subject: Re: USA ruminant-to-ruminant feed ban warning letters ???

 

Date: Mon, 23 Dec 2002 12:56:07 -0600

 

From: "Terry S. Singeltary Sr." Reply-To: Bovine Spongiform Encephalopathy To: BSE-L@uni-karlsruhe.de

 

References:

 

######## Bovine Spongiform Encephalopathy #########

 

Greetings and Happy Holidays,

 

hi Linda, many thanks for this reply, was just checking in to see if anything new had happened since our last correspondence.

 

i thought i had missed something?

 

> Unfortunately, the new database is much more complicated than

 

> the old one, and it does not lend itself to presenting data in

 

> a simple spreadsheet as we did in the past.

 

how convenient;-) i had no problems with the old one...

 

> Please be assured that CVM is working to solve this problem,

 

> and we do plan to post this data in the future.

 

thank you, if USDA/APHIS are lucky, i will hold my breath until that time;-)

 

nothing personal Linda, take care, and may the New Year bring

 

PEACE...

 

TSS

 


 


 

Tuesday, November 04, 2014

 

The pathological and molecular but not clinical phenotypes are maintained after second passage of experimental atypical bovine spongiform encephalopathy in cattle

 


 

*** Singeltary reply ; Molecular, Biochemical and Genetic Characteristics of BSE in Canada Singeltary reply ;

 


 

Tuesday, August 12, 2014

 

MAD COW USDA TSE PRION COVER UP or JUST IGNORANCE, for the record AUGUST 2014

 


 

Thursday, October 02, 2014

 

[Docket No. APHIS-2013-0064] Concurrence With OIE Risk Designations for Bovine Spongiform Encephalopathy

 


 

Saturday, August 14, 2010

 

BSE Case Associated with Prion Protein Gene Mutation (g-h-BSEalabama) and VPSPr PRIONPATHY

 


 

2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006

 


 

10,000,000+ LBS. of PROHIBITED BANNED MAD COW FEED I.E. BLOOD LACED MBM IN COMMERCE USA 2007

 

Date: March 21, 2007 at 2:27 pm PST

 

RECALLS AND FIELD CORRECTIONS: VETERINARY MEDICINES -- CLASS II

 

PRODUCT

 

Bulk cattle feed made with recalled Darling's 85% Blood Meal, Flash Dried, Recall # V-024-2007

 

CODE

 

Cattle feed delivered between 01/12/2007 and 01/26/2007

 

RECALLING FIRM/MANUFACTURER

 

Pfeiffer, Arno, Inc, Greenbush, WI. by conversation on February 5, 2007.

 

Firm initiated recall is ongoing.

 

REASON

 

Blood meal used to make cattle feed was recalled because it was cross- contaminated with prohibited bovine meat and bone meal that had been manufactured on common equipment and labeling did not bear cautionary BSE statement.

 

VOLUME OF PRODUCT IN COMMERCE

 

42,090 lbs.

 

DISTRIBUTION

 

WI

 

___________________________________

 

PRODUCT

 

Custom dairy premix products: MNM ALL PURPOSE Pellet, HILLSIDE/CDL Prot- Buffer Meal, LEE, M.-CLOSE UP PX Pellet, HIGH DESERT/ GHC LACT Meal, TATARKA, M CUST PROT Meal, SUNRIDGE/CDL PROTEIN Blend, LOURENZO, K PVM DAIRY Meal, DOUBLE B DAIRY/GHC LAC Mineral, WEST PIONT/GHC CLOSEUP Mineral, WEST POINT/GHC LACT Meal, JENKS, J/COMPASS PROTEIN Meal, COPPINI - 8# SPECIAL DAIRY Mix, GULICK, L-LACT Meal (Bulk), TRIPLE J - PROTEIN/LACTATION, ROCK CREEK/GHC MILK Mineral, BETTENCOURT/GHC S.SIDE MK-MN, BETTENCOURT #1/GHC MILK MINR, V&C DAIRY/GHC LACT Meal, VEENSTRA, F/GHC LACT Meal, SMUTNY, A- BYPASS ML W/SMARTA, Recall # V-025-2007

 

CODE

 

The firm does not utilize a code - only shipping documentation with commodity and weights identified.

 

RECALLING FIRM/MANUFACTURER

 

Rangen, Inc, Buhl, ID, by letters on February 13 and 14, 2007. Firm initiated recall is complete.

 

REASON

 

Products manufactured from bulk feed containing blood meal that was cross contaminated with prohibited meat and bone meal and the labeling did not bear cautionary BSE statement.

 

VOLUME OF PRODUCT IN COMMERCE

 

9,997,976 lbs.

 

DISTRIBUTION

 

ID and NV

 

END OF ENFORCEMENT REPORT FOR MARCH 21, 2007

 


 

Sunday, December 15, 2013

 

*** FDA PART 589 -- SUBSTANCES PROHIBITED FROM USE IN ANIMAL FOOD OR FEED VIOLATIONS OFFICIAL ACTION INDICATED OIA UPDATE DECEMBER 2013 UPDATE ***

 


 

Tuesday, December 2, 2014

 

UK EXPORTS OF MBM TO WORLD Bovine Spongiform Encephalopathy BSE TSE Prion aka Mad Cow Disease

 

USA, NORTH AMERICA, MBM (or any potential TSE prion disease) EXPORTS TO THE WORLD (?) [protected by the BSE MRR policy] $$$

 


 

Friday, December 5, 2014

 

SPECIAL ALERT The OIE recommends strengthening animal disease surveillance worldwide

 

OIE BSE TSE PRION AKA MAD COW DISEASE ?

 

‘’the silence was deafening’’ ...tss

 


 

Wednesday, December 3, 2014

 

Over 200 Groups Urge Congress to Continue Supporting COOL

 

For Immediate Release

 


 

Monday, December 1, 2014

 

Germany Bovine Spongiform Encephalopathy BSE CJD TSE Prion disease A Review December 1, 2014

 


 

Friday, November 28, 2014

 

BOVINE SPONGIFORM ENCEPHALOPATHY BSE AKA MAD COW DISEASE PORTUGAL CONFIRMED

 


 

Sunday, October 5, 2014

 

France stops BSE testing for Mad Cow Disease

 


 

Monday, May 5, 2014

 

Brazil BSE Mad Cow disease confirmed OIE 02/05/2014

 


 

Sunday, November 23, 2014

 

Confirmed Variant Creutzfeldt-Jakob Disease (variant CJD) Case in Texas in June 2014 confirmed as USA case NOT European

 

*** ‘’The specific overseas country where this patient’s infection occurred is less clear largely because the investigation did not definitely link him to a country where other known vCJD cases likely had been infected.’’

 


 

kind regards, terry